Neurogenin 3 (NEUROG3), the mutation of which can lead to diabetes, is only active for a short time during pancreatic development, which is why its behavior and dynamics, particularly in the context of human development, have been unclear. Researchers at the Max Planck Institute for Molecular Cell Biology and Genetics (MPI-CBG) in Dresden, Germany, and the Novo Nordisk Foundation at the University of Copenhagen have now used a special method to observe both the activity of the gene and the protein it forms in human pancreatic cells to better understand the gene.
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